Possible Mechanisms of Methylmercury Neurotoxicity: Correlation Between the Metabolic Effects of Methylmercury and 5-oxoprolinease Inhibitor on Both C6 Glial Cells and Pc12 - Ezzat Awad - Libros - LAP LAMBERT Academic Publishing - 9783846585504 - 15 de junio de 2012
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Possible Mechanisms of Methylmercury Neurotoxicity: Correlation Between the Metabolic Effects of Methylmercury and 5-oxoprolinease Inhibitor on Both C6 Glial Cells and Pc12

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Different studies reported that glutathione (GSH) play a major role in modulating methylmercury (MeHg) neurotoxicity. Glutathione synthetase deficiency (GSD) and 5- Oxoprolinase deficiencies are two inborn errors of GSH metabolism; which were associated with physiological and pathological features resembling patients with Minamata disease (MD). The present study aimed to investigate whether there are similarities between the metabolic effects of MeHg and 5-oxoprolinase inhibitor (ICA) on both C6 Glial cells and PC12 neurons.

Medios de comunicación Libros     Paperback Book   (Libro con tapa blanda y lomo encolado)
Publicado 15 de junio de 2012
ISBN13 9783846585504
Editores LAP LAMBERT Academic Publishing
Páginas 104
Dimensiones 150 × 6 × 226 mm   ·   173 g
Lengua Alemán